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In this review, we examine the evidence for the emerging paradigm supporting the central role of inflammation rather than specific microbiota in the pathogenesis of periodontitis, and that by controlling the inflammation, it is possible to control the infection.
Abstract. The pathogenesis of periodontitis involves a complex immune/inflammatory cascade that is initiated by the bacteria of the oral biofilm that forms naturally on the teeth.
This review focuses on recent advances in the understanding of the role of endogenous lipid mediators that modulate cellular fate and inflammation. Biosynthesis of eicosanoids and other lipids in exudates coincides with changes in the types of inflammatory cells.
Articles 1–20. Senior Staff Scientist, Johns Hopkins Applied Physics Laboratory - Cited by 735 - Condensed matter theory - Quantum information science.
16 de feb. de 2024 · John Van Dyke via Scopus - Elsevier Recollision Excitation, Electron Correlation, and the Production of High-Momentum Electrons in Double Ionization Physical Review Letters
Historically, inflammatory periodontal diseases (gingivitis and periodontitis) have been recognized as being primarily of bacterial origin. Bacteria are necessary for disease development, but the presence of specific bacteria does not guarantee progression to periodontitis.
Mitigating Errors in DC Magnetometry via Zero-Noise Extrapolation. #1. John S. Van Dyke. , Zackary White. , Gregory Quiroz. ( Feb 26, 2024) e-Print:
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